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Elevated levels of circulating testosterone beginning at puberty in males, on the other hand, tend to predominately affect facial elongation and brow ridge development.
Individuals with higher adult levels of salivary testosterone tend to have superoinferiorly longer faces and more pronounced brows, with brows that are also mediolaterally expanded (with right and left sides tending toward fusing at midline)—a facial morphology that is absent in the masculinized faces of males with low 2D:4D ratios who do not have elevated levels of circulating testosterone (Schaefer et al. 2005).
In males with delayed puberty, treatment with exogenous testosterone has been found to increase upper and total facial height
Figure 1. Craniofacial feminization in Homo sapiens. The 110–90 Ka year old male specimen Skhul 5 (left) in lateral (top) and frontal (bottom) views, compared to that of a recent African male (right), showing the large brow ridges and long and narrow, masculinized face characteristic of MSA/MP-associated modern humans. Both specimens have been scaled to the same nasionbregma height and aligned on those landmarks
This doesn't disprove FWHR being dimorphic and influenced by prenatal testosterone either, as FWHR is specifically about a compact midface, not the facial length overall.